{"id":995738,"date":"2026-08-17T01:03:16","date_gmt":"2026-08-17T05:03:16","guid":{"rendered":"https:\/\/www.marketnewsdesk.com\/index.php\/argenx-announces-positive-topline-results-from-phase-3-alkivia-trial-of-efgartigimod-in-autoimmune-myositis\/"},"modified":"2026-08-17T01:03:16","modified_gmt":"2026-08-17T05:03:16","slug":"argenx-announces-positive-topline-results-from-phase-3-alkivia-trial-of-efgartigimod-in-autoimmune-myositis","status":"publish","type":"post","link":"https:\/\/www.marketnewsdesk.com\/index.php\/argenx-announces-positive-topline-results-from-phase-3-alkivia-trial-of-efgartigimod-in-autoimmune-myositis\/","title":{"rendered":"argenx Announces Positive Topline Results from Phase 3 ALKIVIA Trial of Efgartigimod in Autoimmune Myositis"},"content":{"rendered":"<div class=\"mw_release\">\n<p align=\"center\">\n        \n      <\/p>\n<ul type=\"disc\">\n<li style=\"margin-bottom:6pt\">\n          <i>Study met primary endpoint of mean Total Improvement Score (TIS) at Week 52 in the combined study population of IMNM and DM patients (p=0.0011)<\/i>\n        <\/li>\n<li style=\"margin-bottom:6pt\">\n          <i>Patient improvements observed early and sustained throughout study; consistent treatment effect across IMNM and DM <\/i>\n        <\/li>\n<li style=\"margin-bottom:6pt\">\n          <i>First Phase 3 study to show statistically significant and clinically meaningful improvements in disease activity in\u00a0IMNM, a subtype with no approved therapy <\/i>\n        <\/li>\n<li style=\"margin-bottom:12pt\">\n          <i>argenx to host a conference call on August 17<\/i><br \/>\n          <sup><br \/>\n            <i>th<\/i><br \/>\n          <\/sup><br \/>\n          <i> at 2:30 PM CET\/ 8:30 AM ET<\/i>\n        <\/li>\n<\/ul>\n<p>\n        <b>August 17, 2026, 7:00 AM CET<\/b>\n      <\/p>\n<p align=\"justify\">\n        <b>Amsterdam, the Netherlands<\/b> \u2013 argenx SE (Euronext &amp; Nasdaq: ARGX), a global immunology\u00a0innovation\u00a0company, today announced positive topline results from the ALKIVIA Phase 3 study evaluating VYVGART\u00ae Hytrulo (efgartigimod alfa and hyaluronidase-qvfc) in adults with autoimmune myositis.<\/p>\n<p align=\"justify\">\n        <b>Study met its primary endpoint (p=0.0011)<\/b>\n      <\/p>\n<ul type=\"disc\">\n<li style=\"margin-bottom:8pt;text-align:justify\">In the combined immune-mediated necrotizing myopathy (IMNM) and dermatomyositis (DM) population, patients treated with efgartigimod demonstrated statistically significant and clinically meaningful 15.4-point greater improvement in mean Total Improvement Score (TIS) at Week 52 versus placebo (47.95 vs 32.56).\n<\/li>\n<\/ul>\n<p align=\"justify\">\n        <b>Rapid and sustained treatment benefit<\/b>\n      <\/p>\n<ul type=\"disc\">\n<li style=\"margin-bottom:8pt;text-align:justify\">In the combined population, patients treated with efgartigimod consistently showed improvements over placebo starting at Week 4 that were statistically significant and sustained through the full year of treatment, even with steroid tapering.\n<\/li>\n<\/ul>\n<p align=\"justify\">\n        <b>Magnitude of clinical improvement consistent across both IMNM and DM <\/b>\n      <\/p>\n<ul type=\"disc\">\n<li style=\"margin-bottom:8pt;text-align:justify\">In prespecified subtype analyses, the primary endpoint of Mean TIS at 52 weeks was also met in IMNM patients treated with efgartigimod (p=0.0048), with a 14.8-point greater improvement over placebo (45.05 vs 30.24). In DM, a similar clinically meaningful improvement of 14.5 points (p=0.1093) was observed (51.51 vs 36.96), though statistical significance was not reached in this smaller cohort.\n<\/li>\n<\/ul>\n<p align=\"justify\">\n        <b>Clinical impact observed in both muscle and skin measures<\/b>\n      <\/p>\n<ul type=\"disc\">\n<li style=\"margin-bottom:10pt;text-align:justify\">In both IMNM and DM, all six core set measures of TIS contributed to the treatment effect, each favoring efgartigimod over placebo, spanning muscle strength, everyday physical function, and disease activity beyond the muscle. In DM, improvement in skin disease activity was also observed.\n<\/li>\n<\/ul>\n<p align=\"justify\">Efgartigimod was well-tolerated by patients in the ALKIVIA study. The observed safety profile was consistent with prior studies and the known safety profile of efgartigimod.<\/p>\n<p align=\"justify\">&#8220;For decades, people living with autoimmune myositis have relied on corticosteroids and broad immunosuppression, and those with IMNM have had no approved option at all. These are the first Phase 3 results to show that precision targeting of FcRn with efgartigimod can deliver meaningful benefit in this disease,&#8221; said Luc Truyen, M.D., Ph.D., Chief Medical Officer at argenx. \u201cThe patient response to efgartigimod was durable and multidimensional: separation from placebo emerged early and held through a full year of treatment, with a treatment effect of comparable magnitude in IMNM and DM. This confirms that\u00a0pathogenic IgG autoantibodies are key drivers of autoimmune myositis. We are grateful to the patients, caregivers, and investigators who made this pioneering study\u00a0possible.&#8221;<\/p>\n<p align=\"justify\">\u201cFor people living with myositis, the goal is straightforward: regain strength and function, and get off long-term steroids. Until now we have had limited targeted therapies to offer patients,\u201d said Rohit Aggarwal, M.D., M.S., Professor of Medicine and Co-Director of the Myositis Center at the University of Pittsburgh, and an ALKIVIA investigator. \u201cIMNM is the most refractory form of this disease and many of these patients carry irreversible muscle damage, which makes meaningful improvement genuinely difficult to achieve. That is what makes these results so compelling and groundbreaking. In DM, the magnitude of improvement was comparable \u2013 and for a community where treatment options remain limited and the burden of chronic steroids is just as heavy, that matters. Together, these results tell us that reducing pathogenic autoantibodies is clinically meaningful and a major step forward for patients who are in need of a targeted treatment.\u201d<\/p>\n<p align=\"justify\">Detailed results from the ALKIVIA study will be presented at an upcoming medical meeting.<\/p>\n<p align=\"justify\">Efgartigimod continues to be evaluated as a potential treatment in other autoimmune rheumatologic diseases, including Sj\u00f6gren\u2019s disease and systemic sclerosis.<\/p>\n<p align=\"justify\">\n        <b>argenx Conference Call Details<\/b><br \/>\n        <br \/>argenx will host an investor conference call and webcast today at 2:30 PM CET\/ 8:30 AM ET to discuss the results. A webcast of the conference call may be accessed on the Investors section of the argenx website at\u00a0<a href=\"https:\/\/www.globenewswire.com\/Tracker?data=hDKCMROxqBJkKCEJMRJfM5_4Hex0ESXed9pkLRQT3m3JNruE-fu_CF6t3exSNydLIwIbq5N6_QZi7G3UOWKBP6D2LK1PsSHu4lW4FDV05gGnpF0BdT_VPHD4bvAe4CMFsw7xHZaGMkjehEjNUDlEFtFk4KN3bZKIRFXniOObprVhrvOmDyg3n6wK25Axtd8YHDUaWlDlrOCdMJxQVS4pmD6L0ETnFgcfyJy3P03rufERSvf1-rnqQO7mapr68BzYvDyTWRh4JvbbXzyl4H_41Ouh2oajcZeaLwycVe4SkXgbMUUIcMDzx-jerHktNamMZt4FyP8joWhBtbrBNmfwPDPh6qjv9GOqbc0tnFejWZ7snM-uggTLALGmQGo1pprIhhSNgGWsjPNgfpKGfzdvB2zrKoH5zerYZaUPQzE2wuKTGOupoDQKs_KA7sOtEG2tTqXZe20-acp4RsMVhmkl5aMetCh3t2fFJVUU-e2S4q1hVqADK4cHpr12xKSmn9R7\" rel=\"nofollow\" target=\"_blank\">argenx.com\/investors<\/a>.<\/p>\n<p align=\"justify\">Participants can access the conference call by dialing 800-590-8290 (United States and Canada) or 240-690-8800 (International). Country specific dial-in numbers are listed below:<\/p>\n<p>Belgium\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a032 2290 4635<br \/>France\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a033 172 001717<br \/>Netherlands\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a031 20 795 2683<br \/>United Kingdom\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a044 203 393 1560<br \/>Japan\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a081 3 4520 9761<br \/>Switzerland\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a041 43 210 51 68<\/p>\n<p>\n        <i>Use the access code 3810049 to join the call. Please dial in 15 minutes prior to the live call.<\/i>\n      <\/p>\n<p>A replay of the webcast will be available on the argenx website.<\/p>\n<p align=\"justify\">\n        <b>About the ALKIVIA Study<\/b><br \/>\n        <br \/>The ALKIVIA study was a global, randomized, double-blind, placebo-controlled, multicenter, operationally seamless Phase 2\/3 study of efgartigimod SC for the treatment of autoimmune myositis across IMNM, DM and PM. The ALKIVIA study enrolled 264 patients who had active disease and were on background treatment. Participants were randomized (1:1) to receive weekly injections of efgartigimod PH20 SC or matched to placebo PH20 SC. The study was conducted in two phases, with an analysis of the Phase 2 portion of the clinical trial after the first 89 patients completed the study, followed by Phase 3. The Phase 3 study enrolled 175 patients and included a protocol-mandated corticosteroid taper throughout the study. The primary endpoint of Phase 3 was the mean Total Improvement Score (TIS) at the end of the treatment period of 52 weeks of all treated patients compared to placebo. Prespecified analyses evaluated the combined IMNM and DM population and each subtype separately.<\/p>\n<p align=\"justify\">ALKIVIA was conducted globally across North America, Europe, the Middle East, and Asia-Pacific, including China and Japan. argenx has an exclusive license agreement with Zai Lab for the development and commercialization of VYVGART and VYVGART Hytrulo in Greater China. Through this agreement, Zai Lab recruited Chinese patients into the ALKIVIA trial.<\/p>\n<p align=\"justify\">\n        <b>About Autoimmune Myositis<\/b><br \/>\n        <br \/>Autoimmune myositis is a heterogenous disease spectrum with autoimmune-mediated pathophysiology, characterized by chronic inflammation and progressive muscle weakness, and in some subtypes by skin involvement and other extramuscular manifestations. Proximal muscle weakness is a hallmark clinical feature across subtypes.<\/p>\n<p align=\"justify\">Approximately 100,000 people in the United States live with autoimmune myositis, including approximately 20,000 with IMNM and approximately 40,000 with DM. Up to 80 percent of patients report long-term disability despite treatment. There are currently no targeted treatments available, and care and treatment relies primarily on corticosteroids and broad immunosuppressants, which are associated with significant cumulative toxicity, including metabolic, cardiovascular, musculoskeletal and infectious complications.<\/p>\n<p align=\"justify\">Advances in the understanding of autoimmune myositis biology have highlighted the central role of antibody-mediated immunity, with pathogenic IgG autoantibodies contributing to muscle fiber damage and extramuscular manifestations across subtypes. FcRn maintains circulating IgG levels by recycling IgG antibodies, including pathogenic autoantibodies. Efgartigimod is designed to selectively block FcRn, reducing pathogenic IgG autoantibodies while preserving other aspects of immune function.<\/p>\n<p align=\"justify\">\n        <b>About VYVGART<\/b><br \/>\n        <br \/>VYVGART\u00ae (efgartigimod alfa fcab) is a first-in-class human IgG1 antibody fragment that binds to the neonatal Fc receptor (FcRn), resulting in the reduction of circulating IgG autoantibodies. VYVGART Hytrulo\u00ae is a subcutaneous combination of efgartigimod alfa (VYVGART) and recombinant human hyaluronidase PH20 (rHuPH20), Halozyme\u2019s ENHANZE\u00ae drug delivery technology to facilitate subcutaneous injection delivery of biologics. VYVGART is approved for generalized myasthenia gravis (gMG) and immune thrombocytopenia (Japan only). VYVGART Hytrulo is approved for gMG and chronic inflammatory demyelinating polyneuropathy (CIDP). VYVGART Hytrulo may be marketed under different proprietary names in other regions.<\/p>\n<p align=\"justify\">\n        <b>About argenx<\/b><br \/>\n        <br \/>argenx is a global immunology\u00a0innovation\u00a0company committed to improving the lives of people suffering from severe autoimmune diseases. Partnering with leading academic researchers through its Immunology Innovation Program (IIP), argenx aims to translate immunology breakthroughs into a world-class portfolio of novel antibody-based medicines. argenx developed and is commercializing the first approved neonatal Fc receptor (FcRn) blocker and is evaluating its broad potential in multiple serious autoimmune diseases while advancing several earlier stage experimental medicines within its therapeutic franchises. For more information, visit \u00a0<a 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rel=\"nofollow\" target=\"_blank\">YouTube<\/a>.<\/p>\n<p>\n        <b>This press release contains inside information within the meaning of Article 7(1) of the EU Market Abuse Regulation (Regulation 596\/2014).<\/b>\n      <\/p>\n<p>\n        <b>Media Contact<\/b><br \/>\n        <br \/>Colin McBean<br \/><a href=\"https:\/\/www.globenewswire.com\/Tracker?data=-vWU8Yvth6tBCeL0GaW5mrf0TkUeCeO_-anIL-7B8HUReJDLLCya7uzuiFOZ3uLU3RPe6Vme0q2OXbC0uZNM6vkOIstoxQX9EUJ_GgzI92I=\" rel=\"nofollow\" target=\"_blank\">cmcbean@argenx.com<\/a><\/p>\n<p>\n        <b>Investor Contact<\/b><br \/>\n        <br \/>Alexandra Roy<br \/><a href=\"https:\/\/www.globenewswire.com\/Tracker?data=m52UjVL8ciKnZsYebwV1-xtQkqXHsiB42wpjU3_-cFreJgLwQIQM-BFmxv8nLjfhBApDe6HjbvNGX772W26K0ncb9gNdHYBCPpXQaGLjFGI=\" rel=\"nofollow\" target=\"_blank\">aroy@argenx.com<\/a><\/p>\n<p>\n        <b>Forward-Looking Statements<\/b>\n      <\/p>\n<p align=\"justify\">The contents of this press release include statements that are, or may be deemed to be, \u201cforward-looking statements.\u201d These forward-looking statements generally can be identified by the use of forward-looking words, such as \u201caim\u201d, \u201canticipate\u201d, \u201caspire\u201d, \u201cbelieve\u201d, \u201ccan\u201d, \u201ccontinue\u201d, \u201ccould\u201d, \u201cestimate\u201d, \u201cexpect\u201d, \u201centail\u201d, \u201cforecast\u201d, \u201cfuture\u201d, \u201cgoals\u201d, \u201chope\u201d, \u201cintend\u201d, \u201cis designed to\u201d, \u201clikely\u201d, \u201cmay\u201d, \u201cmight\u201d, \u201cobjective\u201d, \u201cplan\u201d, \u201cpossible\u201d, \u201cpotential\u201d, \u201cpursue\u201d, \u201cproject\u201d, \u201cpredict\u201d, \u201cseek\u201d, \u201cshould\u201d, \u201cstrategy\u201d, \u201ctarget\u201d, \u201cwill\u201d and other words and terms of similar meaning and expression, including in connection with any discussion of future operating or financial performance. By their nature, forward-looking statements involve risks and uncertainties and readers are cautioned that any such forward-looking statements are not guarantees of future performance. argenx\u2019s actual results may differ materially from those predicted by the forward-looking statements as a result of various important factors, including but not limited to, the results of argenx\u2019s clinical trials; uncertainties associated with the development of novel drug therapies; preclinical and clinical trial and product development risks and setbacks; interpretation of argenx\u2019s clinical trial data by regulatory authorities and argenx\u2019s ability to obtain regulatory approval; the risk that early stage clinical trials may not be predictive of results in later stage or large scale clinical trials; the occurrence of adverse safety events or participant dropouts; the acceptance of its products and product candidates by its patients as safe, effective, and cost-effective; the impact of governmental laws and regulations, including tariffs, export controls, sanctions and other regulations on its business; the impact of healthcare regulations, including rules on reimbursement for argenx\u2019s products; competition in drug discovery, development and commercialization efforts; its reliance on third-party suppliers, service providers and manufacturers; and instability and conflicts in the regions in which the company has suppliers or markets for its products. A further list and description of these and other risks, uncertainties, and factors that could cause actual results to differ materially from those referred to in the forward-looking statements can be found in argenx\u2019s U.S. Securities and Exchange Commission (SEC) filings and reports, including in argenx\u2019s most recent annual report on Form 20-F filed with the SEC as well as subsequent filings and reports filed by argenx with the SEC. Given these risks and uncertainties, the reader is advised not to place undue reliance on such forward-looking statements. These forward-looking statements speak only as of the date of publication of this press release. argenx undertakes no obligation to publicly update or revise the information in this press release, including any forward-looking statements, except as may be required by law.<\/p>\n<p>      <img decoding=\"async\" alt=\"\" class=\"__GNW8366DE3E__IMG\" src=\"https:\/\/www.globenewswire.com\/newsroom\/ti?nf=MTAwMTI1NzgyNiM0MDI0ODgzNTcjMjAwMzc3Mg==\" \/><br \/>\n      <br \/>\n      <img decoding=\"async\" alt=\"\" src=\"https:\/\/ml-eu.globenewswire.com\/media\/ZmRmZWY4MWYtMTRkYS00ZTY5LWE4NzUtOWQwMDUwMjljZDEzLTEwMTUzNDUtMjAyNi0wOC0xNy1lbg==\/tiny\/argenx-SE.png\" \/>\n    <\/div>\n<div class=\"mw_contactinfo\"><\/div>\n","protected":false},"excerpt":{"rendered":"<p>Study met primary endpoint of mean Total Improvement Score (TIS) at Week 52 in the combined study population of IMNM and DM patients (p=0.0011) Patient improvements observed early and sustained throughout study; consistent treatment effect across IMNM and DM First Phase 3 study to show statistically significant and clinically meaningful improvements in disease activity in\u00a0IMNM, a subtype with no approved therapy argenx to host a conference call on August 17 th at 2:30 PM CET\/ 8:30 AM ET August 17, 2026, 7:00 AM CET Amsterdam, the Netherlands \u2013 argenx SE (Euronext &amp; Nasdaq: ARGX), a global immunology\u00a0innovation\u00a0company, today announced positive topline results from the ALKIVIA Phase 3 study evaluating VYVGART\u00ae Hytrulo (efgartigimod alfa and hyaluronidase-qvfc) in adults with autoimmune myositis. &hellip; <\/p>\n<p class=\"link-more\"><a href=\"https:\/\/www.marketnewsdesk.com\/index.php\/argenx-announces-positive-topline-results-from-phase-3-alkivia-trial-of-efgartigimod-in-autoimmune-myositis\/\" class=\"more-link\">Continue reading<span class=\"screen-reader-text\"> &#8220;argenx Announces Positive Topline Results from Phase 3 ALKIVIA Trial of Efgartigimod in Autoimmune Myositis&#8221;<\/span><\/a><\/p>\n","protected":false},"author":2,"featured_media":0,"comment_status":"closed","ping_status":"closed","sticky":false,"template":"","format":"standard","meta":{"footnotes":""},"categories":[],"tags":[],"class_list":["post-995738","post","type-post","status-publish","format-standard","hentry"],"yoast_head":"<!-- This site is optimized with the Yoast SEO plugin v28.2 - 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