{"id":751582,"date":"2023-04-26T16:30:48","date_gmt":"2023-04-26T20:30:48","guid":{"rendered":"https:\/\/www.marketnewsdesk.com\/index.php\/autolus-therapeutics-announces-publication-in-molecular-therapy-nucleic-acids\/"},"modified":"2023-04-26T16:30:48","modified_gmt":"2023-04-26T20:30:48","slug":"autolus-therapeutics-announces-publication-in-molecular-therapy-nucleic-acids","status":"publish","type":"post","link":"https:\/\/www.marketnewsdesk.com\/index.php\/autolus-therapeutics-announces-publication-in-molecular-therapy-nucleic-acids\/","title":{"rendered":"Autolus Therapeutics Announces Publication in Molecular Therapy Nucleic Acids"},"content":{"rendered":"

\n– Expanding the Autolus toolkit of T-cell engineering modules against complex and immunologically hostile cancers, including solid cancer applications
\n<\/h2>\n
\n

LONDON, April 26, 2023 (GLOBE NEWSWIRE) — Autolus Therapeutics\u00a0plc<\/a>\u00a0(Nasdaq: AUTL), a clinical-stage biopharmaceutical company developing next-generation programmed T cell therapies, today announced the publication of a paper in Molecular Therapy <\/em>Nucleic Acids<\/em>1<\/sup>, titled \u2018Novel Fas-TNFR chimeras that prevent\u00a0Fas ligand-mediated kill and signal\u00a0synergistically to enhance CAR T-cell efficacy.\u2019<\/em><\/p>\n

CAR T cells have shown remarkable efficacy against hematological cancers, but their effectiveness in solid tumors has been limited by inhibitory factors expressed by the tumor or its microenvironment. One such inhibitory factor is Fas ligand (FasL), which binds to the Fas receptor (CD95) on the surface of an activated T cell and triggers the CAR T cell to die by apoptosis.<\/p>\n

The research group at Autolus tested several Fas chimeras which consist of the extracellular domain of Fas fused to the intracellular domain from different TNF receptor superfamily members. Expression of these chimeras in a CAR T cell not only blocks apoptosis triggered by FasL, but results in co-stimulation, which promotes CAR T cell survival and proliferation.<\/p>\n

A small subset of these chimeras, best exemplified by Fas-CD40 fusion, were found to be particularly effective at promoting CAR T cell survival and anti-tumor cytotoxicity in the presence of FasL. These data support the potential of this Fas-CD40 chimera to render T cell therapies resistant to FasL-mediated cell death and improve their effectiveness against solid tumors.<\/p>\n

\n Martin Pule, Chief Scientific Officer and Founder of Autolus<\/strong>
\n said<\/strong>, “Development of Fas-CD40 adds to our growing toolkit of T-cell engineering modules and aligns with Autolus\u2019 strategy for increasing the Company\u2019s capabilities, with particular application against complex and immunologically hostile cancers, including solid cancer applications.\u201d<\/p>\n

\n 1.\u00a0Novel Fas-TNFR chimeras that prevent\u00a0Fas ligand-mediated kill and signal\u00a0synergistically to enhance CAR T-cell efficacy<\/a><\/strong><\/em>\n <\/p>\n

\n About Autolus Therapeutics plc<\/strong>
\n
Autolus is a clinical-stage biopharmaceutical company developing next-generation, programmed T cell therapies for the treatment of cancer. Using a broad suite of proprietary and modular T cell programming technologies, the Company is engineering precisely targeted, controlled and highly active T cell therapies that are designed to better recognize cancer cells, break down their defense mechanisms and eliminate these cells. Autolus has a pipeline of product candidates in development for the treatment of hematological malignancies and solid tumors. For more information, please visit www.autolus.com.<\/p>\n

\n Forward-Looking Statements<\/strong>
\n
This press release contains forward-looking statements within the meaning of the “safe harbor” provisions of the Private Securities Litigation Reform Act of 1995. Forward-looking statements are statements that are not historical facts, and in some cases can be identified by terms such as “may,” “will,” “could,” “expects,” “plans,” “anticipates,” and “believes.” These statements include, but are not limited to, statements regarding the continued development of Autolus\u2019 AUTO1\/22 program; the status of clinical trials (including, without limitation, expectations regarding the data that is being presented, the expected timing of data releases and development, as well as completion of clinical trials) and development timelines for the Company\u2019s product candidates. Any forward-looking statements are based on management’s current views and assumptions and involve risks and uncertainties that could cause actual results, performance, or events to differ materially from those expressed or implied in such statements. These risks and uncertainties include, but are not limited to, the risks that Autolus\u2019 preclinical or clinical programs do not advance or result in approved products on a timely or cost effective basis or at all; the results of early clinical trials are not always being predictive of future results; the cost, timing, and results of clinical trials; that many product candidates do not become approved drugs on a timely or cost effective basis or at all; the ability to enroll patients in clinical trials; possible safety and efficacy concerns; and the impact of the ongoing COVID-19 pandemic on Autolus\u2019 business. For a discussion of other risks and uncertainties, and other important factors, any of which could cause Autolus\u2019 actual results to differ from those contained in the forward-looking statements, see the section titled “Risk Factors” in Autolus’ Annual Report on Form 20-F filed with the Securities and Exchange Commission on March 7, 2023, as well as discussions of potential risks, uncertainties, and other important factors in Autolus’ subsequent filings with the Securities and Exchange Commission. All information in this press release is as of the date of the release, and Autolus undertakes no obligation to publicly update any forward-looking statement, whether as a result of new information, future events, or otherwise, except as required by law.<\/p>\n

\n Contact: <\/strong>\n <\/p>\n

Julia Wilson
+44 (0) 7818 430877\u00a0
j.wilson@autolus.com<\/a><\/p>\n

Susan A. Noonan
S.A. Noonan Communications
+1-917-513-5303\u00a0
susan@sanoonan.com<\/u><\/a><\/p>\n

Alexandra Deschner
+32-490-58-35-23\u00a0
a.deschner@autolus.com<\/a><\/p>\n


\n
\n \n <\/div>\n

<\/div>\n","protected":false},"excerpt":{"rendered":"

– Expanding the Autolus toolkit of T-cell engineering modules against complex and immunologically hostile cancers, including solid cancer applications LONDON, April 26, 2023 (GLOBE NEWSWIRE) — Autolus Therapeutics\u00a0plc\u00a0(Nasdaq: AUTL), a clinical-stage biopharmaceutical company developing next-generation programmed T cell therapies, today announced the publication of a paper in Molecular Therapy Nucleic Acids1, titled \u2018Novel Fas-TNFR chimeras that prevent\u00a0Fas ligand-mediated kill and signal\u00a0synergistically to enhance CAR T-cell efficacy.\u2019 CAR T cells have shown remarkable efficacy against hematological cancers, but their effectiveness in solid tumors has been limited by inhibitory factors expressed by the tumor or its microenvironment. One such inhibitory factor is Fas ligand (FasL), which binds to the Fas receptor (CD95) on the surface of an activated T cell and triggers … <\/p>\n

Continue reading “Autolus Therapeutics Announces Publication in Molecular Therapy Nucleic Acids”<\/span><\/a><\/p>\n","protected":false},"author":2,"featured_media":0,"comment_status":"closed","ping_status":"closed","sticky":false,"template":"","format":"standard","meta":{"footnotes":""},"categories":[],"tags":[],"class_list":["post-751582","post","type-post","status-publish","format-standard","hentry"],"yoast_head":"\nAutolus Therapeutics Announces Publication in Molecular Therapy Nucleic Acids - Market Newsdesk<\/title>\n<meta name=\"robots\" content=\"index, follow, max-snippet:-1, max-image-preview:large, max-video-preview:-1\" \/>\n<link rel=\"canonical\" href=\"https:\/\/www.marketnewsdesk.com\/index.php\/autolus-therapeutics-announces-publication-in-molecular-therapy-nucleic-acids\/\" \/>\n<meta property=\"og:locale\" content=\"en_US\" \/>\n<meta property=\"og:type\" content=\"article\" \/>\n<meta property=\"og:title\" content=\"Autolus Therapeutics Announces Publication in Molecular Therapy Nucleic Acids - Market Newsdesk\" \/>\n<meta property=\"og:description\" content=\"– Expanding the Autolus toolkit of T-cell engineering modules against complex and immunologically hostile cancers, including solid cancer applications LONDON, April 26, 2023 (GLOBE NEWSWIRE) — Autolus Therapeutics\u00a0plc\u00a0(Nasdaq: AUTL), a clinical-stage biopharmaceutical company developing next-generation programmed T cell therapies, today announced the publication of a paper in Molecular Therapy Nucleic Acids1, titled \u2018Novel Fas-TNFR chimeras that prevent\u00a0Fas ligand-mediated kill and signal\u00a0synergistically to enhance CAR T-cell efficacy.\u2019 CAR T cells have shown remarkable efficacy against hematological cancers, but their effectiveness in solid tumors has been limited by inhibitory factors expressed by the tumor or its microenvironment. 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